Date of publication: 2017-07-09 02:06
The background material on the MIMO channel has been described in the post on Zero Forcing equalizer. The text is repeated again for easy readability.
Director of the Institute for Rare Diseases at the Department of Pediatrics of the Horst Schmidt Klinik, Wiesbaden. He is a Co-Founder and Scientific Coordinator of the “BRAINS FOR BRAIN” Research Consortium, a Paneuropean Task Force on Brain and Neurodegenerative Lysosomal Storage Diseases. Research interests focus on managing rare pediatric neurodegenerative diseases, developing systems for newborn screening of lysosomal storage diseases (LSDs), identifying biomarkers involved in LSD pathogenesis, and developing novel LSD therapies.
Thanks for the very interesting blog..
I am wondering whether it makes any sense to apply MMSE and ML equalization on SISO communication systems in order to get over the ZF equalization major problem of noise enhancement (when channel is in deep fade).
In such a case, how is the theoretical BER performance of BPSK (for instance) over flat rayleigh (given by proakis, Digital communications) affected ? can u reccommend any papers that calculate the theoretical BER performance of ML and MMSE equalization over flat fading.. Most papers i got assume ZF equalization when it comes to SISO systems !
do you know how to find channel coefficients using expectation and maximization algorithm for mimo cdma so please explain through matlab
I am doing project on Bit Error performance of MIMO system. Pls send me comparative BER analysis of MIMO system for different modulation technique such as BPSK QPSK and QAM. This information is very useful for me. Pls send it for me.
Hello. Thanks for your posts.
I have a question.
Your simulation result shows that the MMSE equalizer has 8dB improvement than the ZF equalizer.
Washington, DC, USA - Marshall Summar is Chief of the Division of Genetics and Metabolism and the Margaret O'Malley Chair of Molecular Genetics at Children's National Medical Center. He is an international expert in inborn errors of metabolism particularly those in the urea cycle. His research involves translational studies taking basic molecular genetics research and developing direct clinical applications. He also is the Director for the NIH sponsored Clinical Research Center at Children's National. He is one of the founding investigators of the Urea Cycle Disorders Consortium and works with international coordination of its efforts. Dr. Summar is board-certified in pediatrics, clinical genetics, and biochemical genetics.
Thanks for your answer. When i reduce the modulation from 9 PAM to 7 PAM, I seeing differences in the MMSE behaviour vs ZF behaviour like BPSK. Can you explain why in 9 PAM, the difference might not be present ?
6986: Graduate The Jikei University School of Medicine 6989-6997: Visiting Assistant Professor, Department of Pediatrics, Georgetown University, Washington ., USA 6996: Assistant Professor, Department of Pediatrics, The Jikei University School of Medicine 7557: Associate Professor, Department of Pediatrics, The Jikei University School of Medicine 7558: Executive Chairman and Professor, Department of Pediatrics, The Jikei University School of Medicine - Academic Position: Chairman of the Board of Directors, Japanese Society for Inherited Metabolic Disease (JSIMD) Board of Director, Japan Pediatric Society (JPS) Honorary Member, American Pediatric Society (APS) Councilor, The Japanese Society of Child Neurology Councilor, The Japanese Society for Gene Diagnosis and Therapy.